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| MORE TOWARDS SOLVING A GENETIC MYSTERY IN TYPE-1 DIABETES
(© 13
April 2016: Dr.V.M.Palaniappan, Ph.D.)
Large
number of medical researchers must have read with great interest the
following article that appeared in Medical Xpress (April,11, 2016),
and earlier on in Genes and Immunology.
Even before we can discuss my ADDITIONAL, but EARLIER
input into this area of research, it would make things easier for non-medical
people to understand this very important problem to read the introductory
part of what has been published in Medical Xpress: (http://medicalxpress.com/news/2016-04-genetic-mystery-diabetes.html?utm_source=nwletter&utm_medium=email&utm_content=ctgr-item&utm_campaign=daily-nwletter):
According to Stephan Kissler, Ph.D., Investigator
in the Section on Immunobiology at Joslin Diabetes Center and Assistant
Professor of Medicine at Harvard Medical School (Originally published in: Genes and
Immunology):
In type 1 diabetes, the immune system attacks the
body's own insulin-producing cells.
Scientists don't understand what triggers the attack or
how to stop it
Now the Kissler lab has shown one way in which
one such gene, called RGS1, may help to foster the autoimmune attack.
In the
attack, immune cells called T cells infiltrate the pancreas and
damage the insulin-producing beta cells.
Inhibiting RGS1 didn't prevent autoimmune diabetes from happening … in
humans …
We're continuing to test a number of other genes to see if one strikes
us as being a very potent modifier of type 1 diabetes …
… this piece of information about RGS1 might become valuable down the
line when we know more about other genes…
********
Let me
simplify the entire matter, by avoiding the use of too many medical terms, for
the benefit of our Blog Readers:
For
that matter, even the medical researchers should read this, for the
information contained in this throws light on the possibility of a complete
cure of the problem.
For a
while, let us keep away the intrinsic details concerning the genes and genetics. Let us talk of what we
can understand at ease:
We are
very much aware that there are few types in Diabetes:
1.
Type-1 diabetes mellitus, also called Juvenile Diabetes (in the past)
or Insulin-Dependent Diabetes mellitus.
2.
Type-2 diabetes mellitus, also called Non-insulin dependent diabetes
mellitus.
3.
Diabetes insipidus, characterised by frequent urination.
4.
Pregnancy diabetes, also called Gestational diabetes, and
5.
Drug-induced diabetes.
Certain
cells called Beta cells that are present in Pancreas secrete Insulin hormone.
Insulin
digests sugar (carbohydrates).
If Insulin production is LESS, all
the eaten sugar will not get digested. This results in type-2 diabetes.
To keep
the sugar (carbohydrates) digestion in order, insulin supplements are given
to the patient, in the form of drugs.
If
insulin is given as a supplement from external sources, then there is no need
for the Beta cells in the Pancreas even to try to secrete insulin.
(When
such a thing occurs in the Thyroid gland, that is, if the
thyroxin secretion is NOT enough, then the Thyroid STIMULATING Hormone (TSH) is
secreted. THAT induces the gland to secrete MORE thyroxin.)
When it
comes to diabetes, there is NO such thing as ‘Insulin STIMULATING hormone’.
However,
the sugar WE EAT as part of the food works as Insulin Stimulating Hormone.
In
other words,
If we
eat normal amount of sugar, normal amount of insulin secretes.
If we
REDUCE sugar consumption, Insulin secretion also gets reduced.
If we
INCREASE sugar uptake, insulin secretion INCREASES proportionately.
This is
how the pancreas functions.
***********
Problem
would arise if and when abnormal happenings occur.
Like
everything else, our body has an optimum / best RANGE when it concerns SUGAR
REQUIREMENT.
There
is a minimum and maximum for such sugar requirements.
If and
when we consume MUCH LESS sugar than the MINIMUM, we become totally
energyless, and we can faint, and can even die.
At the
other extreme, if and when we consume TOO MUCH sugar than the MAXIMUM our
body can tolerate, that too can make us die.
If we
consume MUCH LESS sugar, our brain does the following to make us consume MORE
sugar, in order to save us from ultimate death.
It
makes us feel energyless and exhausted.
Our
brain gives us a strong desire to eat sugar – a craving occurs.
When we
eat, the craving stops.
If we OVER-CONSUME
TOO MUCH of sugar, our body does the following, in order to save us from
ultimate death:
To
start with we get the feeling of ENJOYMENT while eating sugar. It is
pleasurable.
Once
the NEED for sugar is over, we FEEL SATISFIED. So, we want to stop eating
more of the sugar.
If we
continued eating sugar, then our brain gives us a feeling of HATE. We hate to
eat more sugar – we get disgusted, in spite of that food being delicious.
At this
stage, let us say some one forces us at gun-point to eat still more sugar.
If and
when we eat more, our brain makes us VOMIT, just to save us from the ultimate
danger of death.
Our
brain, instead of making us vomit, could have simply increased the insulin
production to solve the problem at least as a temporary measure.
In
reality, the brain does that too, but only as a temporary measure.
However,
if we keep on continuing the eating of more and more sugar, and if ALL such
sugars are digested with abundant insulin secretion, then the FAR TOO MUCH OF
THE DIGESTED SUGAR will kill the cells and us, ultimately - the tissues would rot - similar to the GANGRENE that occurs in the long-term diabetic patients with uncontrolled blood sugar levels all the time, necessitating the amputation of the toes or lower part of the leg.
So,
there is a definite need for the brain to totally put a full stop to the
eating of too much of sugar.
The
brain, since time immemorial, from the time of becoming Homo sapiens
from being Apes, or even before that, has associated SWEET TASTE to SUGARS.
In
other words, if SUGAR does NOT TASTE sweet, then our brain will NOT induce
the secretion of Insulin.
The
taste buds in the tongue, the brain, the pancreas, etc. are well
inter-connected, inter-dependent, and remain well-coordinated, helped by SALT
(Sodium) in its capacity as an essential ELECTROLYTE.
Since a
century, Man (not any other animal) has become so intelligent that he has
developed capabilities to HIDE the sweet taste from being felt by the taste
buds in the tongue*.
(*
Similar to adding a likeable perfume to the poisonous mosquito or cockroach
killer poison, the original smell of which is extremely bad and repulsive. We
would avoid inhaling bad smell, whereas we would inhale much deeply if perfumed. That
results in poisoning our brain and body.)
If for
instance, sugar is filled inside CAPSULES, and if the capsules are just
gulped, instead of chewing, then our brain will have no knowledge of the
entry of THAT sugar into the body.
As a
result, brain may not induce insulin secretion, and THIS encapsulated sugar
will NOT get digested.
So, the
undigested sugar will have to keep floating in the body fluids, doing all
possible damages for the well-being of the body*.
(* Swallowing
food without chewing will be similar to consuming capsules.
This is
one of the major reasons why we must chew our food before swallowing.
In
fact, I have recorded that nearly ALL the type-2 diabetic people do NOT
chew their food, but gulp them rapidly: see Palaniappan, V.M., 1998. Obesity:
Causes, Cure, and Prevention. ISBN 967-9988-0508. 471pp.)
In
theory:
If we
happen to consume all sugars only in the form of capsules, or if we SWALLOW
our food rapidly all the time, as described above, our brain will have no
knowledge of the ‘smuggled’ sugar, and thereby loses its management
potentials. In other words, our brain gets deceived*.
As a
result, FAR TOO MUCH OF SUGAR ends up within the body, and disproportionate
insulin quantities leave them to float in the blood in an undigested manner.
This
can bring about the death of the cells and the person, as
described above.
AT
THIS, AN EMERGENCY TO SAVE THE BODY FROM DEATH HAS RISEN!
What
can be done?
The
best possible measure appears to be a TOTAL STOPPAGE OF THE INSULIN
PRODUCTION!
Thus, TWO
emergency situations can warrant instantaneous stoppage of insulin production:
1.
When a person consumes awful lot of sugars (recognisable by the
taste buds in the tongue), almost non-stop, beyond the body’s tolerance
limit, thus inducing the secretion of TOO MUCH of insulin -, beyond the true
need.
2.
When sugar is pumped into the body in a hidden state, essentially due
to gulping the food without chewing*.
Irrespective
of the mode of sugar entry, if there happens to be too much of sugar within
the body, the best way appears to be a TOTAL STOPPAGE of insulin secretion.
If and
when THAT happens, the person gets TYPE- 1 DIABETES.
WE SIMPLY CALL THIS LIFE-SAVING HELP RENDERED BY THE BRAIN AN "AUTO-IMMUNE DISEASE". IF SUCH AN 'AUTO-IMMUNE DISEASE' DOES NOT HAPPEN, WE WOULD DIE! THAT BEING THE CASE, INSTEAD OF CALLING IT AN AUTO-IMMUNE DISEASE, WE SHOULD CALL IT "ALTERNATIVE LIFE SAVING MEASURE", RATHER THAN CALLING IT A DISEASE. ******
In
Type-I diabetes, if the Beta cells in the Pancreas are NOT dead yet (if it is
an early stage), then a REVERSAL should be possible.
In Type
II, where the Beta cells are alive but it is only question of LESSER
insulin secretion, then a REVERSAL appears to be almost DEFINITELY POSSIBLE.
***********
What I
have explained above is all that happens at MACRO-level.
However,
if you want to go academic, and if we are to trace the detailed changes at ‘micro-‘
or even at ‘ultra-micro level’ (similar to seeing detailed cellular
structures through an electron microscope), then of course, we have to
recognise the sequences related to the genes and their interplay.
It
would be even more interesting to see the sequence at ‘Nano’ levels.
Irrespective
of the approach, the result will be the same.
At
macro-level, it would be simple to understand and practise, but will be
unimpressive, and may appear as ‘unscientific’.
The
other academic approach will be extremely impressive, and much complicated to
understand, but will form a necessity for academic excellence. The Ph.D
candidates will have to be trained this way to think and interpret
information much intricately.
Unfortunately,
we do not seem to have much time to wait any further, particularly because
the sufferings and the rate of death due to diabetes alone (besides the
health-care expenditures), appear to be crossing over the bearable limits of
nearly all the nations around the world.
********
We need
to address this issue somewhat ungently.
It
should not matter who does the job. Egoism, professional protectionism, so-called medical ethics, etc. should be kept aloof.
It
should not matter which discipline of medicine helps to achieve success.
All we
need is a successful stoppage of the problem, if possible, all at once.
In the
first place, WHY IS IT THAT ALL THE RESEARCH FINDINGS DONE HITHERTO
WORLD-WIDE DO NOT SEEM TO HAVE THE POTENTIALS TO ERADICATE THE DISEASE?
It
should be because, the APPROACH TO SOLVING THE PROBLEM may not be correct.
If the
CORRECT PROCEDURE is written in a gold plate and is buried in the South Pole,
all the researchers appear to have been digging the soil in the North Pole.
This way, they can never find even after a century - the result can
be chaotic and disastrous.
In
spite of being a non-medical research scientist, I have found the
correct procedure for a total prevention and complete cure of the problem –
Type-2 Diabetes, in particular.
Of
course, my procedure is bound to be VERY HIGHLY CONTROVERSIAL - essentially because of its deviation from prevailing norm.
I can
very easily do identical kind research work similar to all other researchers,
and come up with identical kind of answers, and say that one should do exercises, avoid sugar
consumption, and reduce obesity. This would then CONFORM to what has already been
found by a million scientists around the world. Yet, it would never yield any
beneficial result, except that I will not be criticised!
Is that
my purpose?
I am
not expecting a promotion in my workplace, and therefore, I do not need to be scared
of criticisms. I am
objective-oriented.
I feel
the purpose of my existence is to save mankind at this juncture.
A
workable approach will certainly be unique, unusual, abnormal, and will NOT
conform to the non-working approaches that are being repeatedly said by others.
************
Gunther S. Stent from University of
California in Berkeley calls my kind of report as “PREMATURE DISCOVERY”.
He, along with his colleague Barber, emphasizes
in their paper that discoveries that were
not consistent with the accepted knowledge at the time or not verifiable
technologically would experience the delayed phenomenon.
“delayed recognition papers are
those that are initially unappreciated or unused but are later recognized as
significant”
Dr. Eugene Garfield, Founder of the
Institute for Scientific Information (ISI), who was a pioneer in the field of
citation analysis, and whose data bases have now become the online research
tool of the “Web of Knowledge” (Infoplease, 2007), writes:
“Recognition
is one of the most valued rewards of science. It often is confirmed
exclusively on the individual or team responsible for a particular
breakthrough.
“These
fortunate few certainly deserve the media attention and awards that come with
the success of discovery.”
“It
is almost impossible to identify useful, important, yet unrecognized papers
by any but highly subjective evaluation, but we can recognize a special class
of undervalued papers – those that were recognized long after they were
published. Such papers represent DELAYED RECOGNITION and sometimes are
associated with PREMATURE DISCOVERY.”
“… PREMATURE DISCOVERY IS A SUBJECT OF
DELAYED RECOGNITION.
“A definition, according to Stent, is
that the discovery “was not appreciated in its day.
“This
can occur when the contemporaneous knowledge, technology, and social
issues prevent the discovery from being extended experimentally or
applied to other related scientific efforts.”
Garfield
adds on:
According to William Goffman in
Cleveland’s Case Western Reserve University and Kenneth S. Warren of the
Rockefeller Foundation in New York,
“…A strong presumption prevails that
any evidence that contradicts the accepted view is invalid and must be
disregarded…”
It
seems, Barber had well defined in 1961 itself about the topic of
resistance by scientists to new discoveries (especially those that challenge
commonly held percepts).
Likewise,
if one does not follow the well-ploughed path of presentation of
his/her discoveries, the chances of those findings getting totally ignored
appear to be great.
The
fact that a particular research is done in a deviated manner from the
so-called ‘norm’, should, by right, be considered to show the extra
capabilities and exceptional talent of the researcher.
Again, Eugene Garfield’s (1989) another
thought-provoking article entitled “The Process of Scientific Discovery”
has brought to recognition my kind of approach.
If
all of MY discoveries published in 1998 (and subsequent years) in the
form of a books with proper ISBN reference and the like) is considered to
constitute 100%, recent scientists from different parts of the world have
been publishing these days bits and pieces of my findings (amounting to 5 or
6 percent of my works) as if theirs is the first-time report (may not form
plagiarisms), simply because they have not seen my publications – for want of
adequate citations and publicity, especially because I do not
belong to the mainstream medical fraternity. Thus, my findings tend,
erroneously, to form one of the multiple discoveries.
I have been trying for the past 41 years, through
multivariate means, to get my findings recognized. None of them brought me
any success.
Could you, if you have any authority, kindly evaluate my works, and do the needful
to bring them to recognition soon, so that the suffering millions worldwide
can be saved from nearly all of the diseases, thus making their life on earth
more pleasant, contributive and meaningful.
**********THE WORLD HEALTH ORGANISATION, or any Health Ministry in any of the countries, must come forward to recognise my findings.
FOR THAT MATTER, EVEN OUR MALAYSIAN HEALTH MINISTER Dato Sri Dr. S. Subramaniam, and/or his Deputy Dato Seri Dr.Hilmi Yahaya, who support any alternative medicine if it can help the people rid of their health problems, could invite me to implement my findings in one of our well-established and most modern hospitals in Kuala Lumpur / Selangor (Such as the Selayang Hospital), I will do the needful, demonstrate excellent and highly disciplined scientific procedure and show positive results within a short time span of even TWO MONTHS, incurring insignificant expenditure.
********
Well friends,
I look forward to some magical changes to occur this year.
With best wishes,
Dr. Palani, Ph.D.
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Showing posts with label electrolyte. Show all posts
Showing posts with label electrolyte. Show all posts
Wednesday, April 13, 2016
MORE TOWARDS SOLVING A GENETIC MYSTERY IN TYPE-1 DIABETES
Sunday, July 24, 2011
IODIZED SALT CAN RUIN THE THYROID GLAND IN THIN PEOPLE
IODIZED SALT CAN RUIN
THYROID GLAND
IN THIN PERSONS
Dear Friends,
It appears I have no choice except to making it very long, for if I put in bits and pieces, you may miss the point.
Hence, kindly tolerate, and read it.
Anyway, I will try NOT to post my next article for next few days - thus, giving you time to read leisurely.
Even before we can read the how iodized salt can ruin our health, let us see another useful news related to Thyroid Gland:
I read the news below from the following sources:
(News from US National Library of Medicine, National Institutes of Health, through its information dissemination source: Medicine Plus, which has the following URL, which will be withdrawn after 10/12/2011. http://www.nlm.nih.gov/medlineplus/ news/fullstory _114298.html;
SOURCE: University of Michigan, news release, July 11, 2011; The findings were published online July 11 in the journal Environmental Health Perspectives.
(© HealthDay News by Mary Elizabeth Dallas. http://www.healthday.com/; Dated: July 14, 2011).
EXPOSURE TO COMMON CHEMICALS MAY AFFECT THYROID FUNCTION
In Summary, they have been telling us the following story:
These chemicals are found numerous household products, plastics, solvents, etc.
These chemicals can alter the levels of THYROID HORMONES in the body of people.
If more of the above chemicals enter into the body, lesser will be the hormone productions.
The Thyroid hormone, secreted from the thyroid gland at the throat influence many body functions, including REPRODUCTION and METABOISM.
To find how of these chemicals have gone into the body, normally, the urine samples are tested.
* * * * * *
Click the following URL, and learn more about How to Avoid Bisphenol A and Phthalates
* * * * * *
NOW, LET ME TALK OF MY OWN FINDING THAT SHOWS HOW IODIZED SALT CAN AFFECT OR RUIN OUR THYROID GLAND AND ITS FUNCTION:
(Source: Palaniappan, V.M. Human Diseases: How and Why Do They Occur, and How To Prevent / Correct Them? 2007. 336pp. ISBN978-967-9988-12-3) (It is available in Malaysian bookshops. The recommended price being RM60/-, I am selling it only for RM20/-. Soon, I will try to put it in Amazon.com, if possible).
Under Chapter-6: MISCONCEIVED REMEDIAL MEASURES:
Subheading-13. Iodised Salts
“Reduce salt consumption to the barest minimum, if you cannot totally stop using it.“Consume only iodised salt – and not the natural one.”
These are popular recommendations that are given to every common man in most of the countries. This is a big mistake.
“Take more natural salt, avoid iodised salt, drink 2 L water, and urinate nearly all of it.”
Salt is very important for the well-being of any person. It forms an electrolyte, and helps an individual in many ways.
So, let us now see what I have written under the list of diseases.
It is No. 86 in "Human Diseases" book: “Salt-Caused Diseases”: between pages 228 and 229:
***********************************
Salt-caused Diseases
It appears that excessive salt consumption can thicken the SEMEN, similar to the thickening of the blood.
Salt thickens the blood, and reduces the total volume of the blood, resulting in low B/P initially.
The thick blood should have greater potential to carry more oxygen than normal, resulting in enhanced supply of oxygen to the brain.
If oxygen supply gets reduced for a while (hypoventilation), brain develops dizziness, headache, and even fainting.
Chronic under-supply of oxygen makes the person stupid, and that slows down digestion as well, resulting in making the person obese.
When ample or excessive oxygen is made available (hyperventilation) to the brain, the opposite of all the above can happen.
The brain becomes over-active and food that has already been digested gets rapidly burnt, giving the person frequent hunger
An over-working brain will give rise to insomnia. (Mind keeps on thinking of something.)
This condition, at its extreme, will make the brain work non-stop even if the person under-goes forced sleep with the use of sedatives or sleeping tablets.
As a result of the above, the brain will try to help by inducing frequent urination so that the EXCESSIVE SALT can get removed out of the body through such urinations.
Thus, the person who consumes excessive salt, will be urinating numerous times through the night while trying to sleep.
He will also develop severe thirst for replacement of the body’s lost liquid, and also to help in increasing the quantity of urine.
We can now see the next relevant disease in the listing: i.e., No.44., which talks about Goitre. It can be found between pages 172 and 174, in the “Human Diseases” book:
Please see what I have written in connection with HYPOTHYROIDISM and SALT, under this sub-heading in the same book on Human Diseases:
Goitre (Swelling of Thyroid Gland)
In iodine insufficiency, the thyroid gland swells up, and such a swelling is called goitre.
The non-overweight people, otherwise categorised as the Skeletal and the Thin, normally do not develop hypothyroidism or goitre, for they always take plenty of salt, and thereby their iodine requirement would get satisfied.
However, if the Skeletal, Thin and Perfect people consume the iodine-enriched salt, the iodine would become excessive in their body.
As a result of such an excess, their thyroid gland would secrete excessive thyroxin hormone, giving them HYPERTHYROIDISM, which would make them hyperactive.
Instead, the more food (thereby more iodised salt) they eat, the more will be the hyperactive state of such people.
Any attempt to correct the hyperactivity of the Skeletal people should centre around the elimination of iodine from their body, coupled with the consumption of iodine-free salt.
In addition, the hyperactive people should not consume at all any of the iodine-rich foods such as cabbage, agar-agar jelly, and the ‘sea-foods’ (e.g., sea fish, prawns, etc.).
If the hyperactive Skeletal, Thin or Perfect category people consume far too much of the iodine-enriched salts through their foods and drinks, then, the hypothalamus gland located at the brain would produce too much of a substance called the Thyroid Stimulating Hormone (TSH).
The TSH would induce the Thyroid gland to secrete far too much of the Thyroxin hormone.
Such an abundant Thyroxin will increase hyperactivity in the person to an unusual extreme - beyond his/her body’s tolerance limit - the Threshold Point.
At this, the brain, in order to save him/her from collapsing, induces the hypothalamus gland to secrete another substance, viz., thyroid auto-antibodies, and they are called Thyroglobulin antibody and Microsomal antibody.
As a result, the thyroxin secretion totally stops, leading to the malfunctioning of the body physiology in several ways.
This results in the development of Hashimotos Hypothyroiditis.
THE ABOVE ACTION IS VERY MUCH SIMILAR TO THE WITHDRAWAL OF INSULIN IN THE CASE OF TYPE-1 DIABETICS.
The sufferer is given a daily dose of synthetic Thyroxin hormone, with which he/she can carry on living almost similar to a normal person. There will be no other choice.
However, minor problems related to slight dosage excess or inadequacy are bound to occur every now and then.
Therefore, proper monitoring to decide the right dosage becomes a necessity among this group of people.
Often, all the ill-informed health advocates advise ALL HUMANS to reduce their salt consumption.
The obese people listen to them, and reduce their daily iodised salt consumption.
Whereas, those who are thin continue taking plenty of it in their foods.
In some countries, the governments have made it into a law to force the salt manufacturers to enrich all their products with extra iodine.
When the obese people consume reduced quantities of the iodised salt, their health remains alright.
But, when the thin people take plenty of the iodised salt, they initially become innocent victims of iodine over-consumption that makes them into hyperactive people.
As said earlier, at its extreme, they develop hypothyroidism due to the total dysfunction and death of the thyroid gland.
As per my findings, those who consume more of the natural, UNIODISED salt (to taste) will remain very healthy.
By right, the governments should advocate only the obese people to take less of the iodised salt, while at the same time, make available uniodised natural sea salt for the benefit of the thin people.
One of my VIP patients, who has been taking PLENTY of UNIODISED SALT, told me this true story that had happened while he was with his fellow-golf players:
It seems he was sprinkling awful lot of salt, as usual, over his French Fry. His colleague cautioned him on that.
Instantly, my friend told him, ”My B/P is 129/76, and Pulse is 73, and yours is 160/105, in spite of taking drugs for hypertension, and eating food almost without salt. Further, I am still thin, and you are obese. So, you should not advise me on salt!”
Most of the Japanese people are said to be high-salt consumers. During my short stay in Osaka, I have observed that to be true.
Most such people do not seem to suffer from hypertension.
As I have repeatedly stressed, it is not salt that is the culprit.
The culprits are: NOT TAKING ENOUGH water and NOT URINATING ADEQUATELY!.
I went to India recently, and had a shock.
Do you know why?
I searched and searched in every possible place in the states of Tamil Nadu and Kerala to buy salt that has NOT been iodised.
I could not buy that at all. I tried even through the Chemical Dealers there. After a big search, they apologised to me.
In the end, I HAD to take iodised salt.
Then one thing became very much apparent to me, and that is:
I have studied that ALL “Thin” category people almost definitely take PLENTY of salt.
All Obese people, almost equally definitely take LITTLE salt.
In India, ALL those OBESE people, now taking LITTLE of the IODISED Salt will be relatively much healthier.
However, ALL THIN people, taking TOO much of the IODISED salt, are BOUND to become innocent victims of HYPERTHYROID problems for a while, followed by HYPOTHYROID conditions.
Their thyroid gland may go dead for good.
I am almost very sure that in India, the number of Hypothyroid patients (among the Thin people) must have gone very high up during the past 2 to 3 years - i.e., from the time they made iodising all salts into national-level LAW!
How sad that can be?!
Anyway, if I go to India, I have to take with me some UNIODISED salt packets with me.
That will become handy only if I am going to cook my own food. But, while on travel, I am bound to eat in restaurants….
Can someone help me how to protect myself from the possible dangers of taking IODISED salt?
Calling our friends in India, can you help me?
OK, friends,
I am sorry, today’s presentation had to be this long in order to make the understanding complete.
Kindly tolerate it.
Thanks, and with best wishes,
Dr. Palani.
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