Dr.V.M.Palaniappan, Ph.D.

Featured Post

SEE MY SPECIAL APPROACH FOR AN IMMEDIATE RECOGNITION OF A ‘PREMATURE’ AND BREAKTHROUGH HEALTHSCIENCE DISCOVERY, THAT WAS ‘RESISTED’ ALL THIS WHILE

SEE MY SPECIAL APPROACH FOR AN IMMEDIATE RECOGNITION OF A ‘ PREMATURE ’  AND BREAKTHROUGH  HEALTH SCIENCE  DISCOVERY , THAT ...

Your needs / Objectives / Indemnification

After reading my articles, if you are convinced of their worthiness/ usefulness, you may want to kindly spread the news to your friends suggesting to read what you had read.

My ambition is to reach out to the World Health Organisation, so that my findings will become useful to people worldwide.
'
I will be happy to cooperate / coordinate with any scientist for the furtherance of my findings.

I am extremely THANKFUL to GOOGLE for their fantastic and free services all the time, for reaching out to the public at large.


Indemnification: All my articles are based on MY OWN research, and I strongly believe that they are true. I have been requesting the W.H.O. and Malaysian Ministry of Health to evaluate my discoveries. Until they are approved for use, the Readers of all my articles should get the approval of a Registered Medical Practitioner prior to practising them, and I should not be held responsible for any mishap at all.





With best wishes and thanks,
Dr. Palani, Ph.D.




Ecological Healing System

ALL THE TIME: Popular Posts

Monday, November 3, 2014

GASTRIC / INTESTINAL ULCERS: CAUSES AND CURE, AND A SLIGHT MISHAP THAT HAD HAPPENED IN THE AWARD OF A NOBEL PRIZE.

GASTRIC / INTESTINAL ULCERS: CAUSES AND CURE,
AND A SLIGHT MISHAP THAT HAD HAPPENED
IN THE AWARD OF A NOBEL PRIZE.

(© 2 November 2014: Dr. V.M. Palaniappan, Ph.D.)

Roads at times tend to develop potholes.

Such potholes tend to collect rainwater, often in rainy seasons.

Mosquitoes tend to breed in such stagnant water pools.

A scientist from a popular university collected water samples from the ditches, and examined them at his lab, and found mosquito larvae in them.

Upon finding it, he wrote a paper for publication in the following manner, and submitted it to a popular international journal.

The journal, after subjecting it to ‘severe’ peer-reviewing, published it.

The following is the summary of the paper:

“I examined 20,000 samples, and nearly all the samples had mosquito larvae in them. Therefore, I conclude, for sure, that ALL POTHOLES ARE FORMED BY THE MOSQUITO LARVAE.

Therefore, as a remedial measure, I suggest that all larvae in stagnated pools be killed with some strong insecticides. That will take care of the pothole problems.

All these years, not knowing this ‘fact’, the road-managers worldwide have been ‘erroneously’ filling the potholes with concrete/tar mixtures.

If they can kill the mosquito larvae first, and then fill it with such concrete mixtures, new potholes will not form”.

The above being the ‘latest’ discovery, all news media worldwide, popularized it. Millions of other scientists too referred to this hither-to unknown discovery in their papers.

As a result, THIS paper became the MOST CITED one.

From then on, all road managers in the world have been treating the potholes with insecticides first, and then filling them with a concrete mixture.

How do you like this FICTION? Doesn't it sound silly?

*********
The general belief is, that a most cited paper is considered to contain the best research work. 

Based on such an existing ‘norm’, THIS scientist has every potential to get the year’s NOBEL PRIZE AWARD
 
Would you approve it?

In my opinion, large number of citations or its absence should not be given any significant importance for evaluating the validity of a discovery*

(* My works do not get cited by any of the mainstream medical fraternity in any of their publications, for obvious reasons. My works should not be belittled for any such reasons.)

************
Almost all of us know what is Peptic Ulcer / Gastric Ulcer / Duodenal Ulcer, and the like.

The following is the Press Release from Nobel Committee (on 3 October 2005):

(To get to know the full story please click in the Google search engine the following URL, where you will get a beautiful pictorial illustration of the ulcers as well: 


The Nobel Assembly at Karolinska Institutet has today decided to award the Nobel Prize in Physiology or Medicine for 2005 jointly to Barry J. Marshall and J. Robin Warren for their discovery of "the bacterium  Helicobacter pylori and its role in gastritis and peptic ulcer disease"

This year's Nobel Laureates in Physiology or Medicine made the remarkable and unexpected discovery that inflammation in the stomach (gastritis) as well as ulceration of the stomach or duodenum (peptic ulcer disease) is the result of an infection of the stomach caused by the bacterium  Helicobacter pylori...

.. patients could be cured from their peptic ulcer disease only when the bacteria were eradicated from the stomach. Thanks to the pioneering discovery by Marshall and Warren, peptic ulcer disease is no longer a chronic, frequently disabling condition, but a disease that can be cured by a short regimen of antibiotics and acid secretion inhibitors.’

..Helicobacter pylori ... colonizes the stomach in about 50% of all humans. In countries with high socio-economic standards infection is considerably less common than in developing countries where virtually everyone may be infected.

... In most individuals Helicobacter pylori infection is asymptomatic."

********
In the practice of modern medicine, to pacify the symptom, the patient is treated using drugs*.

(* According to Michael C. DiMarino, MD, has recently (2014) revised the listing 


Drugs for decreasing acidity are used for peptic ulcer, gastroesophageal reflux disease, and many forms of gastritis. Some drugs are used in regimens for treating H. pylori infection. Drugs include:
  • Proton pump inhibitors: They inhibit completely the acid secretion. This is used while treating  H.pylori-caused ulcers.
  • H2 blockers. Proton pump inhibitors form better choice, now, compared to these blockers.
  • Antacids: These agents neutralize gastric acid and reduce pepsin activity (which diminishes as gastric pH rises to > 4.0). 
  • Prostaglandins: this inhibits acid secretion, and  decreases the mucosal injury.
  • Sucralfate: This drug is a sucrose-aluminum complex that dissociates in stomach acid and  forms a  physical barrier over an inflamed area, protecting it from acid, pepsin, and bile salts.)

Upon treating the patient with one or more of the above drugs, his pain – the symptom - would subside.

The victim continues not to eat adequate (or proper) food, and that ‘bad’ habit tends to keep on aggravating the ulceration.

Again, the repeated intake of drugs would, of course, keep on making him forget the pain.

In the meantime, the body’s naturally self-curing potentials help in the regaining of the mucosal lining.

*********
Let ME explain the cause for the development of duodenal ulcer, AS PER MY FINDING:

When a person is HUNGRY (or energyless), the DIGESTIVE JUICE SECRETES to give him an appetite. 
 
The sense of ‘hunger’ is the brain’s language to prompt the person to consume food at once.

At this, if the person refrains from eating any food, naturally, the acid nature of the digestive juice (Hydrochloric Acid + several other acids, including Lactic Acid, Acetic Acid, etc.) tends to erode the mucosal lining of the duodenum/stomach, for that is the location where the juice secretes.

This becomes the pain-giving wound, or ulcer

Since the protective inner lining layer is ‘burnt’ by the acid, the ‘raw’ flesh becomes exposed, and becomes vulnerable for easy penetration of any microorganism.

At this point, the organism Helicobactor pylori, which is freely prevalent, enters as an INVADOR into the tissues, just to ‘eat’ the defenceless flesh as its food. 

As a result, the wound would get aggravated and worsened, and healing would become almost impossible.

Thus, THIS ORGANISM FORMS JUST A SECONDARY INVADER

We call the wound a ‘special niche’ for H. pylori.

In other words, if there is NO wound, there will be NO H. pylori.

(In this sense alone, in the course of treating the patient with the practice of MODERN MEDICINE, it appears is correct to kill the H.pylori using some antibiotics, before the administration of any of the acid suppressants.)

However, unfortunately, the increased intake of the strongly alkaline substance - the antacids, puts up a demand (similar to Biological Oxygen Demand, the BOD) for the secretion of MORE and STRONGER ACID, to cope up the with the neutralizing act. (This happens because the brain does not recognize that the doctor has given this as ‘medicine” with an intention of curing it).

In other words, increased intake of antacids (or, other acid suppressants), tends to proportionately increase the quantity of digestive acids, and also their strength, resulting in the worsening of the ulceration there.

This results in upsetting the body’s natural physiology.
Thus, it becomes a vicious cycle, leading to worsening of the wound, which may even give rise to bleeding, thus necessitating a surgical remedy, at times.

********
How do I go about curing duodenal ulcers, even if they happen to be acute (or chronic), and in an advanced state?

My approach is simple, and is causative:

As said earlier, the digestive juice (with its acid content) secretes when the patient is hungry.

So, the right and easiest approach should be based on ‘satisfying’ the hunger. It is simple as that.

The following regime will ‘cure’ a patient’s ulceration*:
(* If the patient happens to be a diabetic, a I exercise a different kind of action, as per description in my book Diabetes: Causes, Cure, and Prevention, 2011.)

I instruct the patient to consume glucose drinks (made by dissolving 20 grams of dextrose monohydrate in 250 ml of water), three times a day, for three days, all the time with his meals.

For next three days, he has to drink the same, twice daily, once with breakfast, and a second time with his dinner.

He has to drink just one drink with his breakfast for the third set of three days, and then stop it.

This will almost certainly heal his wound.

In the meantime, he will be instructed / trained (as I have described in my "Human Diseases" book mentioned above) to eat MORE food in terms of QUANTITY from then on, even if it happens to be at varying times. (Insisting to consume meals at regular timing does not seem to be necessary for a cure.)

(In the ulcer is in a serious matter, then, the glucose administration will prolong for another one or two weeks, the most).

**********
The following appears to be the basic principle behind the disease occurrence and related treatments I offer:

When a person SPENDS MORE ENERGY (for work + his body functioning) and EATS LESS FOOD, he gets duodenal / gastric ulcer.

When he eats adequate food that equals the energy spent (or more food than necessary), he will NOT get any ulcer at the duodenum.

Increased food that is MORE than his body’s requirement, just for a short term of about two weeks or so (without the administration of any of the antibiotics), heals the wound for good.

This procedure allow's body's natural mechanism to withdraw some of the digestive juice (- the quantity and volume of the acids), thus making drug-induced suppression redundant.

*********
Of course, for obvious reasons, the patient will be told to finish his dinners before 8.00 p.m. the latest. (Lest, he would develop Acid Reflux, and related oesophageal inflammation, along with Antral Gastrities and increasingly unbearable pain, and the like. You may want to read one of my earlier postings on Curative Procedure of Acid Reflex disease, in this Blog).

Patients treated this way do not get back their ulcerations or related pains.

(When it comes to treating to cure a patient suffering from advanced ANTRAL GASTRITIS, although the basic principle remains the same, the treatment pattern differs, with extra suggestions to change the composition of his diet, along with changes in the of dinner timings.)

In both cases, surgical procedure appears to be altogether redundant.

I have successfully cured with a lot of ease a large number of patients (? about a hundred, so far) suffering from such problems.

I have established that administration of antacids or any kind of ANTIBIOTICS to KILL H.pilori is totally unwarranted for a cure of duodenal ulcer (or even Antral Gastritis).

Let us recollect from the Nobel Committee’s press release (shown above) the following points of interest:
  • That this disease is the result of infection of the stomach caused by the bacterium Helicobacter pylori... 
  • That H. Pylori is found in about (only) 50% of all humans.
  • This infection is severe in poor countries. 
  • That in most cases, the H.pylori infection does NOT create any PAIN.

The above statements show:
  • If the disease can occur in 50% of the patients who do NOT have H.pylori infection, then, H. pylori cannot be the causative for the disease. 
  • The severity of the infection is high in poor countries because, large number of people do not consume ENOUGH food to provide them with enough of the required ENERGY. That is, they are under-fed, and overworked. This keeps them in a constant state of unsatisfied hunger. That is what gives them this disease, and  NOT H.pylori
  • In rich countries, people eat enough (or more) food, and therefore, the prevalence of this disease is very much less. This shows again, that the disease arises essentially because of hunger, and not because of the infection by H.pylori
  • In most cases, H. pylori remains asymptomatic, and does NOT give rise to pain. Such a state shows that H.pylori has absolutely nothing to do with the creation of ulcer as such – it is only a secondary invader, with some potentials for increasing the distress*, and that should be all to it. 
(* It is analogous to the following: if a patient’s foot is injured by a nail in the shoes he wore, not only that nail can give him the pain, but ANY  OTHER thing such as little stone on the road or a tiny stick too can give identical pain. So, such a pain-giver should not be considered as a causative matter for the presence of wound in the foot of the sufferer.)

*********
In, what I wish to highlight is:
  • It is, in the first place, it is wrong to accept the statement that it is H. pylori that is CAUSING the peptic ulcer. 
  • Likewise, it is also wrong to treat the patient with antibiotics, unnecessarily. 
  • Administration of antacids and acid-suppressants in the conventional way of treating these ulcers should be considered redundant, especially because of the side and after effects. 
  • Since the aetiology is directly related to shortage of food energy, the patients should be told to eat more food for a cure, or, if a need arises, he should be provided with food by philanthropists or International Agencies created for such purposes. 
  • The press release of the Nobel Committee may require some amount of re-phrasing so that it will reflect the corrected understanding, rather than leaving it as it is, thus giving room for my kind of criticism. 
  • According to Root Bernstein (The Sciences, 1988:28:26-34), any mental activity that contributes directly to scientific discoveries should be recognized as scientific method.
  • The Nobel Assembly at Karolinska Institutet should evaluate ALL of my findings (found in my book The True Causes of All Diseases) for a possible award of the Prize, at least for 2015 – may be before I die, for they do give it posthumously. (I do not feel ashamed to ask for it, for I know the worthiness of my findings.)

One of my 20 books, entitled “Human Diseases: Why and How Do They Occur, and How to Prevent / Cure Them?” (ISBN 978-967-9988–12-3) (2007. pp 152-156) has little more information on stomach ulcers and natural cures.

I have explained in the above book as why and how even rich people who can afford to have plenty of food develop gastric problems of this kind. 

Correcting their diseases without drugs would require only a change in their lifestyle.

Dear Friends,
I thank you for reading such a long paper.
With best wishes,

Dr. Palani, Ph.D.