Dr.V.M.Palaniappan, Ph.D.

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SEE MY SPECIAL APPROACH FOR AN IMMEDIATE RECOGNITION OF A ‘PREMATURE’ AND BREAKTHROUGH HEALTHSCIENCE DISCOVERY, THAT WAS ‘RESISTED’ ALL THIS WHILE

SEE MY SPECIAL APPROACH FOR AN IMMEDIATE RECOGNITION OF A ‘ PREMATURE ’  AND BREAKTHROUGH  HEALTH SCIENCE  DISCOVERY , THAT ...

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My ambition is to reach out to the World Health Organisation, so that my findings will become useful to people worldwide.
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I am extremely THANKFUL to GOOGLE for their fantastic and free services all the time, for reaching out to the public at large.


Indemnification: All my articles are based on MY OWN research, and I strongly believe that they are true. I have been requesting the W.H.O. and Malaysian Ministry of Health to evaluate my discoveries. Until they are approved for use, the Readers of all my articles should get the approval of a Registered Medical Practitioner prior to practising them, and I should not be held responsible for any mishap at all.





With best wishes and thanks,
Dr. Palani, Ph.D.




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Showing posts with label BRCA1. Show all posts
Showing posts with label BRCA1. Show all posts

Sunday, March 4, 2012

New Gene Change is linked to Breast Cancer

NEW GENE CHANGE IS LINKED TO BREAST CANCER

(© 4 March, 2012: Dr. V.M. Palaniappan, Ph.D.)

The following news appeared in HealthDay, on February 22, 2012:
Researchers Spot New Gene Mutation Linked to Breast Cancer.

Since you may find it a bit difficult to understand the original text, let me simplify the statement:

Scientists already know that there is a gene called BRCA1, and that is called a Breast Cancer Gene.

There is another gene called Abraxas Gene.

This Abraxas Gene drastically changes (i.e., mutates) for ‘some’ reason, and interacts with the Breast Cancer Gene.

This is said to make genetic alterations that increases the breast cancer risk.

This research was done by Associate Professor of Cancer Biology Dr. Roger Greenberg and his colleagues at Perelman School of Medicine in University of Pennsylvania, USA.

This was originally published in the journal called Science Translational Medicine. You can read the full text through this source: http://stm.sciencemag.org/content/4/122/122ra23.full

MY INTERPRETATION:

Let us say that you had a small cut on one of your fingertips. It bleeds, then stops after clotting.

You may feel the pain. However, if you took care of the wound so that it does not come in contact with water and the like, and keep it dry, it would heal in a day or two, provided that you are not a diabetic patient.

The above has been occurring since time immemorial.

Nowadays, with the advancement of science, and a need for academicians to publish research papers for a promotion in their career, they go much deeper to explain every little thing in much greater detail.

Thus, in reality, if you refer to some medical books for details of the wound that was found in your finger tip, they would have published ultra-microscopic view of the fresh wound to start with, then the changes that are found during the natural healing process.

They would also assign complicated names in Latin or Greek language to describe them.

A commoner would get perplexed by all the above.

However, if you are look at the overall picture, there was a bleeding cut initially, then it healed, and some of the tissues dried up and came off from the healed spot. That was all to it!

Likewise, if you are to look at the BREAST CANCER, you have two ways of interpreting it:

As a scientist (e.g., Oncologist - cancer specialist) intense details such saying “The breast cancer gene, viz., BRCA1”, etc., you can simply describe in the following manner,  of course based on MY STUDY and FINDINGS:

Some tissues in the breast are designed to absorb calcium from lymph and blood fluids. Using this calcium, the breast produces milk for to feed the baby.

If breast milk is fed to the baby, the scavenged calcium gets out of the breast tissues.

In the absence of breast feeding, the calcium tends to accumulate and form a benign lump.

If and when more and more calcium gets absorbed, the lump becomes much bigger in its size, and eventually turns cancerous.

Why should this calcium accumulate in a woman who has not delivered any baby? Often, it occurs even in virgins.

The answer is:
 
Under normal circumstances, the excess calcium brought to the tissues in the breast gets removed through frequent urinations.

If and when a woman avoids urination for any reason and withholds the urinal pressure, the excessive calcium, since it does not get removed, stays in the breast tissues, 
accumulates more and more, and eventually turns into a lump, and then into a malignant state.

(When urine is withheld, the water part alone gets out of the body in the form of profuse sweat, in a manner I have explained in our previous postings in this Blog as well as in my books.)

The calcium excess will increase in quantity, and within shorter duration, if the girl happens to take calcium supplements, calcium-enriched food items, and calcium-rich anchovies, and the like.

Thus, to the victim, in layman’s term, the simplified story will be:

“A girl did not urinate properly, she also consumed foods and supplements that contained abundant calcium in larger quantities, and as a result of these, she developed breast lump initially, and that turned out to be cancerous subsequently”

Of course, the above story can also be explained using scientific terminology and in intense detail, with electron-microscopic views of the many stages, as well.

Please note: The above explanation of the risk factor for breast cancer and related details are my own findings, and does NOT form an explanation of the original news about the mutation of the gene and its linkage to the breast cancer found by Professor Roger Greenberg and his colleagues.

OK, friends, bye for the time being,
Dr. Palani, Ph.D.

Monday, February 6, 2012

WHY SHOULD OBESE MEN WITH PROSTATE CANCER FACE HIGHER DEATH RISK


WHY SHOULD OBESE MEN 
WITH PROSTATE CANCER 
FACE HIGHER DEATH RISK.

(©: 6 February 2012: Dr.V.M.Palaniappan, Ph.D.) 
A study finds it that the obese men with prostate cancer face higher death risk. It finds 2.6 fold increase in chance of dying compared to normal weight men (Hutchinson (2007; ScoutNews, 2007).
  
   My Interpretation (as is presented in my cancer book):
 
   Needless to highlight that all soft tissues (including the prostate gland and heart) of all obese men, in the first place, are bound to become enlarged (inflammation) because of the absorption of free calcium (in the absence of sunshine or vitamin-D) due to under-urination and chronic constipation.
 
  Whereas such an initial enlargement would not occur in the thin men. (They are thin because they urinate 7 or more number of times daily, through which nearly all the calcium excesses get out of the body, thus not contributing to blood vessel thickening (arteriosclerosis), heart blockages, prostate enlargement, or kidney stones.)
 
   In other words, because of the calcium absorption, the prostate gland of obese men can be assumed to enlarge, say '2.6 fold’, when the thin persons have just “0” fold enlargement (i.e., no enlargement at all).
 
  With the above starting points, IF both groups of men consume, for example, twice daily, soups* made of vegetables containing abundant calcium, the obese men would certainly cross over the prostate’s threshold point (i.e., tolerance limit) much earlier than the thin men. Hence, the disparity in the death rate.

   (* Soups are made by boiling vegetable, chicken, crabs, or any such thing in water. During this process, most of the calcium contained in those items (calcium, being water-soluble) would get extracted into the soup. Thus, soups form concentrated calcium-containing liquids.)
 
  In the obese, the major reason for an earlier death should be essentially due to their reduced water consumption and under-urinations habits, which thicken their blood vessels and create blockages in the heart.   
 
*     *     *     *     *     *
In USA, prostate cancer appears to be affecting black men more often than the white or Hispanic men (Hoffman et al, 2001).
 
  My Interpretation (as is found in my cancer book):
 
  I am not too sure if the African Americans habitually consume more calcium containing eatables and drinks.
 
  Besides the eating habit, the African American men c(as well as women) could be under-urinating all the time*.

   ( * This may have some relevance to the past life style of the African Americans - in the past, a few generations of them were forced to work almost non-stop all the time. They had to control their urge to urinate then and there, and thereby the whole lot of them became big-sized, due to the retention of the calcium excesses within their body. Such an under-urinating could have become 'norm' for all of them along with time. Hence, they could be more susceptible to obesity, and obesity-related diseases, including prostate cancer.)
 
   In other words, the African Americans, as of today, should be under-urinating all the time*.

  (* A study to trace this important phenomenon, by tracing the 'Liquid (Water Intake) / Urine Output' should be conducted to recognise this fact. In good health, the Ratio should be 1.0, and can go up to 1.2.  However, I won't be surprised if it goes to 1.5 or more. I think, it may even go up further to, even 2.0. Any reading above 1.2 would give rise to HYPERHIDROSIS (profuse sweating in palms, feet, and all over the body), and would also make the person OVERWEIGHT AND OBESE, ending up as MORBILDY OBESE.)
 
  This makes them ‘giant-sized’ (Palaniappan, 1998) due to calcium accumulation within the cells of all the organs and muscles in the body. 
 
   As a result of the above two phenomenon, the prevalence of prostate cancer among the black men could be more than the white or Hispanic men, as reported by Hoffman et al, in 2001. 
 
*     *     *     *     *     *
Men who have a brother or father with prostate cancer have twice the usual risk of developing prostate cancer (Steinberg et al, 1990).
 
   My Interpretation (as is found in my cancer book):
 
   I have explained repeatedly in several of my books/publications that members of the same family could easily end up developing identical eating / water drinking habits.
 
   Generally, people all over the world are being told to consume more green leafy vegetables for maintaining good health.
 
   Following this, most of the health-conscious housekeepers tend to prepare soups of vegetables that contain abundant calcium, on a daily basis.
 
   As a result of this, all members of the same family tend to consume such ‘rich’ soups almost daily. They could even become addicted to this soup-drinking habit.
(I have often come across several Chinese men in Malaysia getting ‘addicted’ to soup-drinking.

The moment they enter into a restaurant, the first thing they order is, of course, a big bowl of vegetable soup.

Prostate cancer appears to be more prevalent among Chinese men in Malaysia.
 

This should be seen from the angle of the Theory of Evolution as explained by Darwin, and in relation to the  “Selection of the Best” and  “Survival of the Fittest”.

Only the best and good characters and features will get transmitted to the next generation, and NOT the worse and diseases!)
 
   As a result of this, there several male members of the same family could develop prostate enlargement and cancers.

   An obese mother, who is obese by virtue of NOT drinking enough water and NOT urinating adequately, would NOT encourage her children to drink water, and may not train her infants to urinate liberally. This would end up making her children too obese. This would then pose to scientists as it that trait is an inherited / genetic factor. Apparently, that would only constitute an error.
 
  Therefore, this should not be mistaken as some sort of genetic involvement.
 
*     *     *     *     *     *
Two genes (called BRCA1 and BRCA2) that are important risk factors for ovarian cancer and breast cancer in women have also been implicated in prostate cancer (Struewing et al, 1997).
 
   My Interpretation (as is found in my cancer book):
 
  We have to consider two different aspects here:
 
(1) The two genes are said to be common to all the three cancers.
 
   This supports my finding clearly that the root cause for the development of cancers is the accumulation of calcium within the soft cells. Hence, the commonness among these three.
 
   This being the truth, I won’t be surprised if Geneticists come up with more findings to prove that several more cancers have identical risk factors.
 
(2)  To the best of my knowledge, no one can visually ‘see’ or ‘read’ the risk details in the genes.
 
   They should be matters of only interpretations of certain criteria.
 
   Therefore, I am of the opinion that the so-called genes BRCA1, 2, etc. could be only indications of some factors present in the chromosomes / body of the person, and not of any direct relationship to deteriorating characteristics.
   
   References:

   Palaniappan, V.M. 1998. Obesity: Causes, Cure, and Prevention. 471pp. Ecohealth Sdn.Bhd. ISBN 978-967-9988-05-8.

    Palaniappan, V.M. 2008. The True Causes of All Diseases. 190pp. ISBN 978-967-9988-13-0).

   Palaniappan, V.M. 2010. Cancer: Causes, cure and prevention. Neo Health Care. 610 pp. ISBN 978-967-9988-14-7.
*********
Dear Friends,
Please keep reading these postings. I will soon be presenting some amazing information you wouldn't have expected...
With best wishes,
Dr. Palani, Ph.D.