Dr.V.M.Palaniappan, Ph.D.

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SEE MY SPECIAL APPROACH FOR AN IMMEDIATE RECOGNITION OF A ‘PREMATURE’ AND BREAKTHROUGH HEALTHSCIENCE DISCOVERY, THAT WAS ‘RESISTED’ ALL THIS WHILE

SEE MY SPECIAL APPROACH FOR AN IMMEDIATE RECOGNITION OF A ‘ PREMATURE ’  AND BREAKTHROUGH  HEALTH SCIENCE  DISCOVERY , THAT ...

Your needs / Objectives / Indemnification

After reading my articles, if you are convinced of their worthiness/ usefulness, you may want to kindly spread the news to your friends suggesting to read what you had read.

My ambition is to reach out to the World Health Organisation, so that my findings will become useful to people worldwide.
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I will be happy to cooperate / coordinate with any scientist for the furtherance of my findings.

I am extremely THANKFUL to GOOGLE for their fantastic and free services all the time, for reaching out to the public at large.


Indemnification: All my articles are based on MY OWN research, and I strongly believe that they are true. I have been requesting the W.H.O. and Malaysian Ministry of Health to evaluate my discoveries. Until they are approved for use, the Readers of all my articles should get the approval of a Registered Medical Practitioner prior to practising them, and I should not be held responsible for any mishap at all.





With best wishes and thanks,
Dr. Palani, Ph.D.




Ecological Healing System

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Wednesday, February 15, 2012

EXCESSIVE USE OF MULTIVITAMINS CAN INCREASE PROSTATE CANCER



1. CAN THE EXCESSIVE USE OF MULTIVITAMINS
 INCREASE PROSTATE CANCER RISK? 
  ( 15 February, 2012: Dr.V.M.Palaniappan, Ph.D.)

  According to a new study by NIH, USA:

  There have found the existence of some evidence suggesting that Vitamin-E and Selenium can protect against prostate cancer. 

At the same time, they have also found that excessive use of the supplements can increase the risk . 
     
   In a 2007 National Institute of Health Study, men who took multivitamin supplements more than seven times a week increased the risks for developing advanced prostate cancer and for dying from the disease.
     
The risks were highest for men who had a family history of prostate cancer and for those who took individual supplements of selenium, beta-carotene, or zinc (HealthTalk, 2008).

(HealthTalk, 2008. Porostate cancer. New guidelines for localised prostate cancer. http://www2.healthtalk. com/go/reports/?p=10/000033.htm)
  My Interpretation:

In the first place, as per my study, Prostate Cancer is not genetically linked, and it cannot be inherited / transmitted from parents to children (Palaniappan, V.M., 2010. Cancer: Causes, Cure and Prevention)

I have established that it is calcium that enters into the prostate gland to give rise to its enlargement first, and then cancer there (Palaniappan, 2010).

I very much doubt if vitamin-E and/or selenium can have any effect in reducing the prostate cancer. Even Zinc might not have had any such effect them.

However, the second statement given above says that the risk increased in those who took either excessive of it, or as individual supplements.

The latter can be true only if either one of the above had the capacity to somehow scavenge to bring in calcium into the soft cells of the Prostate Gland. Again, I am unable to comprehend the existence of such a possibility.

What I am not sure of is that, these scientists who did the study did not know that it is Calcium EXCESS that gives cancers. Therefore, there is every possibility that they have inadvertently included Calcium as a part of the supplement while administering the vitamin-E, selenium and/or zinc for the testing, with the assumption that Calcium has nothing to do with cancer, and has everything to do only with the bones and teeth.

Anyway, I think, I may have explore the full paper a little more to understand what is being conveyed in that news release.
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2. HORMONE THERAPY FOR PROSTATE CANCER  
   Hormone Therapy is said to have shown little benefit against prostate cancer (Yeo, 2008).
  
   My Interpretation: 
   I have already established that it is calcium accumulation in the cells of the prostate gland that gives rise to initial enlargement and subsequent cancer there (Palaniappan, 1998-2010).
    
  This being the case, the female sex hormone oestrogen may not have any negative effect on the development of prostate cancer.

However, there could be an effect for the following reason:

If the female hormone oestrogen suppressed the production of semen, then, there is the possibility that the calcium which is normally utilised in the making of the semen was left unused. In such case, the freed calcium can get into the soft cells of the prostate gland itself to enlarge its size, and eventually to create a cancer there. But then, this should be considered as a positive relationship between the two, rather than negative.

This is so because, the oestrogen (the female hormone used in the therapy) suppresses a man’s desire to have sex. As discussed earlier in one of my articles in this Blog, I have explained as how any reduction in seminal discharge would only increase calcium accumulation at the prostate, and would speed up the development of prostate cancer there.

     On the other hand, if a man is given extra  androgen hormone his vigour and desire for sex would increase.

When he has frequent sexual unions, then, all the calcium excesses would get out of his body through the seminal fluids, thus keeping him in a healthy state. If this happens, the Prostate gland will not enlarge, and a cancer may not form.

      In all obese men, influenced by the accumulation of       excessive calcium, their androgen production gets much reduced, and thereby their desire for sex as well as their seminal fluid discharges get drastically reduced.

Reduced androgen secretion tends to reduce sperm count. As a result, the calcium, for want of ‘storage space’, would enter into the few sperms, and  that would make them 'obese' - hypertrophy occurs (- sperms becoming bigger size is not good).

Such bigger sized and enlarged sperms tend to lose their power for motility, and hence they fail to fertilise a woman during sexual unions.

That is one of the reasons why most of the obese men remain sterile (Palaniappan, 2007).

I wish to recollect here that the trial in which some researchers tried giving the female hormone oestrogen to men to find if that had any effect in reducing the heart attacks in men, based on the belief that it is oestrogen that has been protecting all the menstruating women from getting any heart attack (until they get their menopause).

I have proven otherwise: the female hormone as such has nothing to do with the prevention of heart attacks in menstruating women.

In fact, it is the regular loss of calcium in the menstrual fluids at monthly intervals that saves them from such myocardial infarctions. 

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OK, friends, will come up with another write-up soon.
Bye, 
Dr. Palani, Ph.D.